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Hope for Back Pain

At times, it may seem to the back pain sufferer that there is little or no hope for ever being "normal" again. Some readers may say to themselves, "If I have to go through all this every day of my life, I might just as well have the whole thing fused surgically and live with a stiff low back."

I would strongly argue that this is the wrong attitude. Surgery should only be viewed as the very last resort for unremittant, debilitating, pain or vertebral instability when neurological function is compromised or at risk of being lost. Later in the website, you will learn that, even for people who undergo a fusion surgery or discectomy, the predisposition for additional degenerative disc disease still exists at other (especially adjacent) levels of the spine. Without intentional intervention, the damaging forces acting upon the discs will still be present and capable of inducing further future disability and pain. Quite often, the surgery is only a partial discectomy and the same disc continues to degenerate. Just because you have one disc repaired, doesn't mean that your problems with your spine are solved. Quite the contrary, if you don't alter the mechanics, there is every reason to believe that other discs will fall prey to the same forces that damaged the original one.

In fact, my observation has been that an inordinately large percentage of persons with Lumbar disc disease eventually present with similar Cervical or Thoracic disc problems and visa versa. This leads me to conclude that there must be certain genetic predispositions to faulty disc mechanics and that the ramifications of some inherent structural protein difference results in a weakness of ligamentous capacity that is reflected in one person's ability to sustain the same amount of force without damage to the disc whereas another person under identical circumstances ends up with a damaged disc. I suspect it has something to do with the tensile strength and elasticity of their collagen fibers (the proteins that compose ligaments and cartilage) and that there are certain genetic subsets of persons who are destined, by virtue of their hyper-elastic collagen, to have an increased probability of disc disease regardless of whether or not they sustain major, forceful injuries. This trait may make them evolutionarily more likely to survive by giving them the "wirey" capacity to wriggle free of their enemies; but the gains they achieve in elasticity leave them deficient in tensile strength. If this is true, these people (of which I think persons with hyper-flexible joints and/or scoliosis may be an extremely affected subset) may be prevented from what otherwise appears to be an inevitable fate. However, it is too early for me to make that speculation formally; and this website is not the appropriate forum. Suffice it to say, I have enough information to advance the suspicion and hypothesis because I have observed scoliosis induced solely by disc disease. Time and wide-spread use of The O'Connor Technique (tm) will determine whether this suspicion is correct. Until then, persons with early scoliosis are free to make and act upon the assumption that the origin of their disease process rests in hyper-migratory disc material and use The O'Connor Technique (tm) to try to prevent disfiguration. They certainly are unlikely to come to any additional harm by practicing these techniques; and I would enjoy learning if they appear to be successful, so that a comprehensive, scientific study could be rapidly assembled to test that hypothesis. The number of scoliosis patients I currently, or ever, will see in my practice is so small as to be negligible--someone else will have to study that question.

Contrary to political rhetoric, all men are not created equal. However, that is not to say a person with a predisposition for a bad back is inferior to someone with an intact spine because having extra-flexible collagen may impart some other selection advantage and survival value. Judging from the multitudes with bad backs alive today, it seemingly doesn't carry any Darwinian selection disadvantages. Perhaps the increased flexibility carries with it an, as yet unrecognized, selection advantage, the usefulness of which becomes less significant after offspring are successfully reared.

However, it does not follow that simply because one has a bad back that it is the result of an inexorable genetic failing. One needn't adopt the fatalistic attitude that their Fate is immutable or that they are pre-destined to suffer or I would not have written this treatise.

Hope does exist in the peculiar capacity the human organism has for accommodation on an intellectual and structural level. Understand that acting upon knowledge can favorably change fate, and the body is not a static entity. It is constantly being broken down and rebuilt on a microscopic level. Although, to the unenlightened, when viewing a skeleton it seems to exist as a hard, rigid, structural frame that is unchanging in life as much as it is in death. Quite the contrary, even this rigid bony structure is constantly being broken down and reformed at the cellular level. This rebuilding can be modified depending upon the stresses applied. Obviously, there are certain structures that, once broken, cannot be rebuilt to their original functional capacity. There are certain conditional restrictions upon the ability of the human organism to repair damage. Yet, over time, the pre-programmed capacity of the body to modify its structure can be capitalized upon to expect eventual healing and return to a near-normal functional activity level.

A major constituent of The O'Connor Technique (tm) is a fundamental methodology that optimizes the capacity of the human body to intellectually and physically adapt to spinal damage as well as maximize effective repair. These components are referred to as FLEXION AVOIDANCE, MAINTENANCE OF EXTENSION, DYNAMIC POSITIONING, and PREFERENTIAL STRENGTHENING/SELECTIVE HYPERTROPHY. With these and other innovations in back pain management, The O'Connor Technique (tm) has taken a great step forward in reducing the healing time, decreasing the duration of back pain events, and preventing or reducing the frequency of future episodes. Separate sections in the website specifically address these components and throughout the website wherever pertinent, they are reiterated in contexts where appropriate. The reader who understands and practices them can expect to have true hope for a largely pain-free future.

1. Bigos SJ, Deyo RA, Romanowski TS, Whitten RR, The new thinking on low back pain, Patient Care; July 15, 1995:140-172

2. Bigos S, Bowyer O, Braen G, et al. Acute Low Back Problems in Adults. Clinical Practice Guideline, Quick Reference Guide Number 14. Rockville, MD: U.S. Dept. of Health and Human Services, Public Health Service, Agency for Health Care Policy and Research, AHCPR Pub. No. 95-0643. December 1994.

3. Korff, et al., Pain , 1988, 32:173-83.

4. Reis S, et al., Low Back Pain: More than Anatomy, The Journal of Family Practice, 1992;35(5):509.

5. Deyo RA, Mayer TG, et al., The painful back: Keep it moving, Patient Care, October 30, 1987:47-59.

6. Frymoyer JW, Helping your patients avoid low back pain, The Journal of Musculoskeletal Medicine, May 1989:83-101.

7. Frymoyer JW, Helping your patients avoid low back pain, The Journal of Musculoskeletal Medicine, May 1989:83.

8. Graves EJ. Diagnosis-Related Groups Using Data from the National Hospital Discharge Survey: United States--1985. Hyattsville, Md: 1987. US Dept of Health and Human Services publication 87-1250.

9. Miller S, Total Fitness Program Best for Low Back Problems, Family Practice News, June 1-14, 1989; 19(11):37.

10. Op.Cit. Endnote #1;p.140-172

11. Cole HM (ed.), Diagnostic and Therapeutic Technology Assessment (DATTA), The Journal of the American Medical Association, Sept 19, 1990, 264, (11):1469--1472.

12. Sack B, Acute and Chronic low back pain: How to pinpoint its cause and make the diagnosis, Modern Medicine, Sept. 1992;(60):58-92.

13. Vlok GJ, Hendrix MR. The Lumbar disc: evaluating the cause of pain. Orthopedics, 1991; 14:419-25.

14. Deyo RA, Loeser JD, Bigos SJ. Herniated Lumbar intervertebral disk. Ann Intern Med, 1990;112:598-603.

15. Predicting Outcome in Back Pain Patients, Family Practice News, Oct 1-14, 1986:1-36.

16. Frymoyer JW. Back pain and sciatica. N Engl J Med. 1988;31;82-91

17. Anon, Family Practice News, August 1-14, 1987.

18. Anon, Back Pain Is a Definitive Diagnosis Necessary?, Emergency Medicine; February 15, 1993:131-134.

19. Cherkin D, Deyo RA, Berg AO, et al, University of Washington, Seattle, and other centers; Evaluation of a physician education intervention to improve primary care for low-back pain I: Impact on physicians. Spine, Oct 1991;16:1168-1172.

20. Cherkin D, Deyo RA, Berg AO, et al, University of Washington, Seattle, and other centers; Evaluation of a physician education intervention to improve primary care for low-back pain I: Impact on patients. Spine, Oct 1991;16:1173-1178.

21. Kriyananda, Yoga Postures for Self-Awareness,(1969) Ananda Publications, San Francisco, CA, USA.

22. Yogiraj Sri Swami Satchidananda, Integral Yoga Hatha, (1970), Holt, Rinehart and Winston, New York, USA, p. 49.

23. McKenzie, R, Treat Your Own Back, Spinal Publications LTD., P.O. Box 93, Maikanae, New Zealand, p.9.

24. Mooney V, quoted in Backache: What Patterns of Pain Pinpoint the Source?, Data Centrum, April 1984;1(4):25-37.

25. Weber H, Lumbar disc herniation: a controlled, prospective study with tens years of observation. Spine. 1983;8:131-140.

26. Saal JA, Saal JS, Herzog RJ. The natural history of Lumbar intervertebral disc extrusions treated non-operatively. Spine, 1990;15:683-6.

27. Williams PC. Low back and neck pain causes and conservative treatment.2nd ed, Springfield, Ill:Charles C Thomas, Publisher;1974.

28. Leblan K, et al. Report of the Quebec task force on spinal disorders. Spine. 1987;12:S1-S8.28.

29. Op.cit., Bigos, Endnote #2.

30. Donelson RG, Identifying appropriate exercises for your low back pain patient. The Journal of Musculoskeletal Medicine; December 1991:14-29.

31. Weed D, Chiropractic Care, The Specialists Neck and Shoulder Injuries Lecture, August 31, 1995, Fairfield, CA.

32. Ross EC, Parnianpour M, Martin D, The effect of resistance level on muscle coordination patterns and movement profile during trunk extension. Spine. 1993;18:1829-1838.

33. Flicker PL, Fleckenstein JL, Ferry K, et al. Lumbar muscle usage in chronic low back pain. Spine. 1993;18:582.

34. Valkenburg HA, Haanen HCM. The epidemiology of low back pain, in White AAIII, Gordon SL (eds) American Academy of Orthopedic Surgeons Symposium on Idiopathic Low Back Pain, St. Louis, CV Mosby Co, 1982:9-22.

35. Troup JD, Martin JW, Lloyd DC, Back pain in industry: A Prospective Survey, Spine (1981) 6:61-69.

36. Cherkin D, MacCornack FA. Patient evaluation of low back pain care from family physicians and chiropractors. West J Med, 1989, 150:351-5.

37. Hockberger RS, Meeting the Challenge of Low Back Pain, Emergency Medicine; August 15, 1990:99-127.

38. Associated Press, Report on the causes of stroke fingers chiropractors, San Francisco Sunday Examiner and Chronicle, 2/20/94:A5.

Article Contents:
You are not alone
The Pain
Contemporary Perspective on Back Pain
Historical Perspective of Back Pain
Science and Art
Alternative Therapeutic Modalities
Back Surgery
Comparative Programs
Not an Excercise Program
Dismissing the "Psychological" Contribution To Spinal Pain
Getting Better as a Process
Become your own Chiropractor
Hope

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MAKING YOUR BAD BACK BETTER, with The O'Connor Technique™, How You Can Become Your Own Chiropractor, by William Thomas O'Connor, Jr., M.D.
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ISBN:
0-9664991-1-5
Publication Date: 02/01/2000
Publisher Name: AEGIS GENOMICS CORPORATION
Price: $37.95
Format: Paperback
Pages: 402
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